Shear Stress - The Left Main and Coronary Artery Bifurcation Lesion Summit (CBS)

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Shear Stress

Abstract

Flow-Regulated Endothelial S1P Receptor-1 Signaling Sustains Vascular Development
TAVI Represents an Anti-Inflammatory Therapy via Reduction of Shear Stress Induced, Piezo-1-Mediated Monocyte Activation
Low shear stress induces vascular eNOS uncoupling via autophagy-mediated eNOS phosphorylation
Coronary Microcirculation in Ischemic Heart Disease
Evolving understanding of the heterogeneous natural history of individual coronary artery plaques and the role of local endothelial shear stress
Low shear stress induces endothelial reactive oxygen species via the AT1R/eNOS/NO pathway
High Coronary Shear Stress in Patients With Coronary Artery Disease Predicts Myocardial Infarction
Local Low Shear Stress and Endothelial Dysfunction in Patients With Nonobstructive Coronary Atherosclerosis
Endothelial ACKR3 drives atherosclerosis by promoting immune cell adhesion to vascular endothelium

Original Research

JOURNAL：Circulation.

TAVI Represents an Anti-Inflammatory Therapy via Reduction of Shear Stress Induced, Piezo-1-Mediated Monocyte Activation

S Baratchi, MTK Zaldivia, M Wallert et al.

Keywords: Piezo-1; TAVI; aortic valve stenosis; high shear stress; low shear stress

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