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Pulmonary Hypertension Caused by a Coconut Left Atrium ACCF/AHA 2009 expert consensus document on pulmonary hypertension a report of the American College of Cardiology Foundation Task Force on Expert Consensus Documents and the American Heart Association developed in collaboration with the American College of Chest Physicians; American Thoracic Society, Inc.; and the Pulmonary Hypertension Association Pulmonary hypertension related to congenital heart disease: a call for action Survival prospects of treatment naïve patients with Eisenmenger: a systematic review of the literature and report of own experience Medical Therapy for CTEPH: Is There Still Space for More? A Case of Pulmonary Hypertension Associated with Idiopathic Hypereosinophilic Syndrome Pulmonary Artery Denervation Using Catheter based Ultrasonic Energy 2019 ESC Guidelines for the diagnosis and management of acute pulmonary embolism developed in collaboration with the European Respiratory Society (ERS): The Task Force for the diagnosis and management of acute pulmonary embolism of the European Society of Cardiology (ESC) Diagnosis and management of acute deep vein thrombosis: a joint consensus document from the European Society of Cardiology working groups of aorta and peripheral vascular diseases and pulmonary circulation and right ventricular function A prediction model of simple echocardiographic variables to screen for potentially correctable shunts in adult patients with pulmonary arterial hypertension associated with atrial septal defects: a cross-sectional study
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Clinical Case StudyAvailable online 23 April 2020

JOURNAL：Can J Cardiol. Article Link

Pulmonary Hypertension Caused by a Coconut Left Atrium

EC Leung, N Hirani et al. Keywords: pulmonary hypertention; coconut left atrium; chest x-ray image

ABSTRACT

“Coconut atrium” was first described in 1898 and is most commonly associated with rheumatic heart disease. This case demonstrates a dramatic chest x-ray image of extensive circumferential calcification of the left atrium. The abnormality was thought to result in elevated pulmonary pressures and severe right heart failure in the absence of significant mitral valve dysfunction.
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